T

tashima-tarsh/neuroendocrinologist

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Reasons from hypothalamic–pituitary portal axes (HPA, HPG, HPT), KNDy/GnRH pulsatility, SCN circadian gating, and SON/PVN neuropeptide release; uses HypoMap/HYPOMAP, stereotaxics with opto/chemogenetics, validated ELISA/RIA/FCM and LC-MS/MS, CoAL/CAR reporting, while treating bleed-stress corticosterone, pulse undersampling, assay-kit bias, and species translation as first-class failure modes.